Vocab #1 Vocab #2 Cellular Adaptation Cellular, reversible/nonreversible Stress Response
100
Cells shrink and reduce their differentiated functions in response to normal and injurious factors
Atrophy
100
Irreversible damage resulting in cellular death.
Cellular Necrosis
100
Three stages; Alarm, Resistance/Adaptation, and Exhaustion.
What is General Adaptation Syndrome (GAS)
200
Increase in cell mass accompanied by an augmented functional capacity in response to physiologic and pathophysiologic demands
Hypertrophy
200
First manifestation of most reversible cell injury, cellular swelling due to the accumulation.
Hydropic Swelling
200
The point where the body can no longer return to homeostasis.
What is exhaustion
300
Increase in functional capacity related to an increase in cell number due to mitotic division
Hyperplasia
300
Programmed cellular death.
Apoptosis
300
Agents or conditions that can produce stress; endanger homeostasis.
What are stressors
400
Replacement of one differentiated cell type with another
Metaplasia
400
4 types of Necrosis
Coagulative, Liquefactive, Fat Necrosis, Caseous Necrosis
400
Constricts blood vessels and raises blood pressure, reduces gastric secretions, and increases night and far vision.
What is norepinepherine
500
Disorganized appearance of cells because of abnormal variations in size, shape, and arrangement
Dysplasia
500
Cellular injury resulting from oxygen deprivation.
Hypoxia
500
Primary glucocorticoid, affects protein metabolism, promotes appetite and food-seeking behaviors, and has anti-inflammatory effects.
What is cortisol






Patho Week 1

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