Arterial blood pressure is directly proportional to cardiac output and this.

Systolic pressures > 180mmHg

These agents seek to relive blood pressure by inhibiting normal vasoconstriction mechanisms in the smooth muscles of arterioles and veins.

These agents work in the collecting tubule and duct and prevent K+ loss and excrete Na+ followed by water loss.

This product of the RAAS is a powerful vasoconstrictor.

Baroreflexes and the Renin-Angiotensin-Aldosterone System (RAAS)

The corresponding systolic pressure of stage 2 hypertension.

These drugs encourage salty pee by supressing the synthesis of angiotensin II and suppressing aldosterone.

These drugs inhibit the Na/K/Cl cotransport inthe ascending Henle loop resulting in retention of Na, Cl, and water in the tubule.

This protein is released from the kidney in response to a drop in blood pressure or fluid volume.

Primary, essential, malignant, resistant.

A normal diastolic pressure should be less than this number.

This is the target site of the "artens" class of drugs. Losarten, valsarten, candesartan, and so on.

These drugs inhibit resorption of Na and Cl in the distal convoluted tube. They are the most often used agent of this.

This is released from the liver to react with renin from the kidney.

This form of HTN has no cure and treatment is to lessen symptoms and improve life quality.

Elevated systolic pressure is in this range.

Though the mechanisms by which they work vary, the action of this class of drugs all share the same target area and seek to lower blood volume thus decreasing blood pressure.

Less effective than thiazides or loop diuretics, these agents work in the proximal convoluted tube.

This end product of the RAAS acts on the kidneys to retain NaCl and water.

This lonely type of hypertension corresponds to ventricular contractions and can produce pressures near 200mmHg.

Heart disease, heart failure, stroke, and chronic kidney disease.

A helpful Pneumonic to remember the varied MOA of this class of drugs is, "A through N" and "O through Z".

This class of drugs blocks Na and K exchange, keeping Na in collecting duct at the cortical level.

This enzyme is released from the lungs to react with another enzyme to form a powerful vasoconstrictor.